Frontiers MicroRNA-34c Inhibits Osteogenic Differentiation and Valvular Interstitial Cell Calcification via STC1-Mediated JNK Pathway in Calcific Aortic Valve Disease
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Calcific aortic valve disease (CAVD), a common heart valve disease, is increasingly prevalent worldwide and causes high morbidity and mortality. Here, we aim
Frontiers MicroRNA-34c Inhibits Osteogenic Differentiation and Valvular Interstitial Cell Calcification via STC1-Mediated JNK Pathway in Calcific Aortic Valve Disease
The expression of STC1 is downregulated by miR-34c. (A) intersection
Midkine Prevents Calcification of Aortic Valve Interstitial Cells via Intercellular Crosstalk. - Abstract - Europe PMC
Frontiers Identifying hub genes of calcific aortic valve disease and revealing the immune infiltration landscape based on multiple WGCNA and single-cell sequence analysis
Novel mechanisms for osteogenic differentiation of human aortic valve interstitial cells - ScienceDirect
Frontiers NOTCH Signaling in Aortic Valve Development and Calcific Aortic Valve Disease
Valve Interstitial Cell-Specific Cyclooxygenase-1 Associated With
MiR-34c plays a role in CAVD via mediating JNK pathway by inhibiting
MicroRNA-22 promoted osteogenic differentiation of valvular interstitial cells by inhibiting CAB39 expression during aortic valve calcification
Histone deacetylase 6 reduction promotes aortic valve calcification via an endoplasmic reticulum stress-mediated osteogenic pathway - ScienceDirect
Human Stanniocalcin-1 Blocks TNF-α–Induced Monolayer Permeability
Abstract 15010: Inhibition of MicroRNA-34a Attenuates Osteogenic Differentiation via Inhibiting Notch1 Signal in Aortic Valve Interstitial Cells
Calcification Induced by Type I Interferon in Human Aortic Valve Interstitial Cells Is Larger in Males and Blunted by a Janus Kinase Inhibitor
Role of Oxidative Stress in Calcific Aortic Valve Disease: From Bench to Bedside - The Role of a Stem Cell Niche
MicroRNAs regulate signaling pathways in osteogenic differentiation of mesenchymal stem cells (Review)